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What are COVID shots doing to your blood?

According to standard narrative, clotting events after 'vaccination' with spike-protein factories not linked to shots

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Y Rabinovitz

Y Rabinovitz

Y Rabinovitz

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March 29, 2022

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01:53 AM

What are COVID shots doing to your blood?

A young British woman, just 34 years old, complained of an excruciating headache eight days after being vaccinated against COVID. Within hours she was dead. According to the coroner, Mrs. Kim Lockwood had been “extremely unlucky”.

This was, after all, in March of 2021 when less was known about the effects of spike protein in the body. Lockwood’s death (following the AstraZeneca shot) was attributed to Vaccine-Induced Thrombotic Thrombocytopenia (VITT) but “medical advances” since the vaccines were introduced meant that the condition was “better recognized” (according to the BBC).

Better recognized – but not better treated. The coroner noted that an MRI scan should have been performed sooner, but admitted that nothing could have saved the young mother’s life due to the massive “sudden and catastrophic” brain hemorrhage she suffered.

The specific problems with the AstraZeneca shot and blot clotting were more-or-less understood by the end of 2021 as detailed, to give just one example, in the Science Advances online journal. 

Vaccines derived from chimpanzee adenovirus ... [can cause] ultrarare side effects not seen in phase 3 trials, including thrombosis with thrombocytopenia syndrome (TTS), a rare condition resembling heparin-induced thrombocytopenia (HIT) ... This study demonstrates that all three adenoviruses deployed as vaccination vectors versus SARS-CoV-2 bind to platelet factor 4 (PF4), a protein implicated in the pathogenesis of HIT. (emphasis added)

Blood clotting was recognized very early on as one of the more dangerous developments that could result from COVID infection. According to some studies as many as 30 percent of strokes and deaths in COVID patients were caused by blood clots. It took a little while longer for the virus’ spike protein specifically to be implicated, but as early as August, 2020, researchers from India noted in an article published in Frontiers in Physiology that,

The nucleocapsid and spike proteins of the SARS-CoV-2 have been speculated to contribute to a pro-thrombotic state due to their modulation of clotting pathways in the lungs via dysregulation of ACE2…” (emphases added).

Throughout 2021 researchers were starting to take more notice, as a study published in Bioscience Reports in May of that year illustrates.

SARS-Cov-2-induced infection ... is characterized by unprecedented clinical pathologies. One of the most important pathologies, is hypercoagulation and microclots in the lungs of patients. Here we study the effect of isolated SARS-CoV-2 spike protein S1 subunit as potential inflammagen sui generis. Using scanning electron and fluorescence microscopy as well as mass spectrometry, we investigate the potential of this inflammagen to interact with platelets and fibrin(ogen) directly to cause blood hypercoagulation. Using platelet poor plasma (PPP), we show that spike protein may interfere with blood flow. Mass spectrometry also showed that when spike protein S1 is added to healthy PPP, it results in structural changes to β and γ fibrin(ogen), complement 3, and prothrombin ... Here we suggest that, in part, the presence of spike protein in circulation may contribute to the hypercoagulation in COVID-19 positive patients and may cause substantial impairment of fibrinolysis. Such lytic impairment may result in the persistent large microclots we have noted here and previously in plasma samples of COVID-19 patients. This observation may have important clinical relevance in the treatment of hypercoagulability in COVID-19 patients. (emphases added)

A few months later, in August, 2021, an article in Cardiovascular Diabetology added some more puzzle pieces to the picture.

“SARS-Cov-2-induced infection ... may be accompanied by hypercoagulation and platelet hyperactivation...”

The study set out, however, not to investigate the role of the spike protein in causing clots, but rather whether the COVID infection itself was the cause of potentially fatal microclots. Spike protein was mentioned only once in the entire article, where the researchers wrote, 

Mass spectrometry was performed using a Thermo Scientific Fusion mass spectrometer equipped with a Nanospray Flex ionization source. Plasma samples, before and after addition of spike protein addition (1 ng mL−1 final exposure concentration), from4 of our control samples were analysed with this method. (emphases added).

By this time – late summer of 2021 – mass vaccination had been well underway in many countries, even though the mRNA shots had only received emergency use authorization, meaning that they were still within their trial period. And yet, researchers were focusing not on the properties of the shots themselves (miniature spike protein blueprints) but rather on the COVID infection that was supposedly already taken care of via those shots. (That turned out to be a major disappointment, but that is not the topic at hand.)

How could researchers use spike protein to imitate COVID infection and investigate that infection, and simultaneously remain blind to the obvious significance of the results they were seeing for the COVID shots themselves? Continuing from the same study,

COVID-19 survivors complain of recurring fatigue or muscle weakness, being out of breath, sleep difficulties, and anxiety or depression. Given that blood clots can block microcapillaries and thereby inhibit oxygen exchange, we here investigate if the lingering symptoms that individuals with Long COVID/PASC manifest might be due to the presence of persistent circulating plasma microclots that are resistant to fibrinolysis.

Conclusion: Clotting pathologies in both acute COVID-19 infection and in Long COVID/PASC might benefit from following a regime of continued anticlotting therapy to support the fibrinolytic system function. (emphases added)

The conclusion, moreover, partially obscures one of the main problems noted, namely, that the plasma microclots formed by the spike protein were found to be “resistant to fibrinolysis".

Fibrinolysis is a process that prevents blood clots from growing and becoming problematic. Primary fibrinolysis is a normal body process, while secondary fibrinolysis is the breakdown of clots due to a medicine, a medical disorder, or some other cause.

Would the anticlotting therapy the researchers suggest help with these resistant clots? The coroner cited at the start of this article, presumably informed by expert medical opinion, conceded that it would not have in the case of the 34-year-old British woman. She, of course, is just one of many. How rare are such cases? Not so rare that most of us haven’t heard of several.

The article concludes by connecting the dots between endotheliopathy and “critical illness and death". Endotheliopathy was linked to spike protein as far back as 2020 in several studies, including some that suggested that this was a way the virus could find its way into the brain.

The cognitive dissonance continued throughout 2021. In October, an article published in bioRxiv started from the almost incredible position that, 

Blood clots are a central feature of coronavirus disease-2019 (COVID-19) and can culminate in pulmonary embolism, stroke, and sudden death. However, it is not known how abnormal blood clots form in COVID-19.… (emphases added)

The researchers then reported the rather dated finding that,

Here we report that the Spike protein from severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) binds to the blood coagulation factor fibrinogen and induces structurally abnormal blood clots with heightened proinflammatory activity ... One-sentence summary: SARS-CoV-2 spike induces structurally abnormal blood clots and thromboinflammation.... (emphases added)

Early in 2022, America’s Frontline Doctors received an intriguing letter.

My name is Richard Hirschman, I am a mortician in Alabama. I have been embalming for 20 years, and I have been noticing strange clotting. I am not sure exactly when it began but probably around June of 2021. I know what blood clotting is and over 20 years I have seen plenty of them, but these are not the same. They have a strange substance in them, looks like fibers. I call it ‘the worms,’ because sometimes they look like worms. I took a picture of one in late September because people would not believe me. I have been taking photos of others as well, so far I have taken over 30 pictures all are from different bodies. Since November I have been trying to make notes on bodies with the clots. I would say that over 50 percent of the bodies have them. I have spoken to other embalmers and they are seeing the same thing.

Hirschman then continued:

I am afraid that it’s vaccine connected but I can't prove it. I usually do not know if the person was vaccinated or not but I do know that some are. I noticed an increase of clotting since the beginning of the Covid pandemic but the clots didn't have the ’worms.’ I wish I knew what the substance is and what's causing it. I am feeling the need to share what I am seeing, if it is the vaccine causing this people need to be warned. I will attach some photos and you can request more if you want.

(Hirschman did attach photos to his letter which are available upon request from AFLDS.)

When Hirschman first spoke out, his was a lone voice. As is so often the case, other voices then joined his. One was that of Anna Foster, who contacted Steve Kirsch:

Anna Foster is an embalmer with 11 years of experience in Carrollton, MO. The big news is that she found the unusual clots in 93% of the last 30 people who she embalmed. This is significant because she isn’t selective about who she embalms.

Not all embalmers will see a 93% clot rate. Richard Hirschman only sees these clots in 65% of his cases. Her embalmer friends have noticed it as well and have never seen it before in their careers.

The clots are only associated with people who have been vaccinated. They were only observed after the vaccines rolled out. The clots are life threatening and are almost certainly the root cause of death in all of these cases.

Steve Kirsch also interviewed Dr. Ryan Cole, who explained the process of clot formation in the presence of spike protein and noted that it has been reproduced in laboratory conditions. 

Some of the clots found by Hirschman, Foster, and others, are over ten inches long. Some extend the entire length of a human leg. They were found by embalmers who attempted to inject the special fluids necessary to preserve the body and were unable to do so, until these clots were manually removed.

If you ask the Centers for Disease Control (CDC), they’ll tell you this is impossible. 

According to the CDC,

The mRNA and the spike protein don’t last long in the body.

Our cells break down mRNA and get rid of it within a few days after vaccination.

Scientists estimate that the spike protein, like other proteins our bodies create, may stay in the body up to a few weeks.

That means, according to the standard narrative, that strange clotting events that occur weeks after “vaccination” with spike-protein factories are not linked to the shots.

But a study from Stanford University has debunked that. Published in Cell at the end of January, 2022, the study found that,

... vaccine mRNA and spike protein persist in lymph nodes for up to two months following the second vaccine dose. This is in contrast to what happens following infection, where spike protein was found only rarely...

At least some portion of spike antigen generated after administration of BNT162b2 [Pfizer shot] becomes distributed in the blood...

The high concentration in the blood of spike protein following vaccination and its persistence along with vaccine mRNA in lymph nodes for months, in contrast to the situation post-infection where such persistence is rare, will fuel concerns about the safety of these COVID vaccines. It has been argued that the spike protein is itself pathogenic, not inert, and that the free spike proteins generated by the vaccines have greater capacity to bind to more types of cells than the virus particles themselves, and that this may be what lies behind many of the serious adverse events reported to regulatory bodies and identified in case reports. This warrants further investigation. (emphases added)

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